By R.E. Hester, R.M. Harrison
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Additional info for AIR POLLUTION & HEALTH
S. Linn, D. A. Shamoo, C. E. , Environ. , 1983, 30, 340. R. A. Bethel, D. J. Erle, J. , Am. Rev. Respir. , 1983, 128, 592. J. R. Balmes, J. M. Fine and D. Sheppard, Am. Rev. Respir. , 1987, 136, 1117. D. Horstman, L. J. Roger, H. , Toxicol. Ind. Health, 1986, 2, 289. 5 job:LAY01 21-9-1998 page:6 colour:1 black–text J. G. Ayres Figure 1 Distribution of individual airway sensitivity to SO as cumulative percentage of 27 subjects. PC(SO ) is the provocative concentration of SO required to increase airway resistance by 20% dioxide (Figure 1) with a median PC sRaw of 750 ppb (2145 g m\).
There are no satisfactory longitudinal studies which have considered the effects of gaseous pollutants like the Six Cities Study and the American Cancer Association Study have considered the effects of particulate pollution in this regard. One series of studies of Seventh Day Adventists (an attractive study group as these individuals do not smoke cigarettes, thus removing the major complicating cause of respiratory and heart disease) has suggested that long-term exposure to ozone is associated, in men only, with an increased risk of developing asthma.
Unfortunately, these different approaches mean that it is not always possible to undertake comparisons between studies. Because most of the studies from the US and Canada have been undertaken in the summer, the main pollutants assessed have been particles, ozone and aerosol strong acid (the ozone studies being discussed below), SO being regarded until lately by North American workers as an unimportant pollutant for their countries, largely on the basis of low measured ambient levels. A. S.
AIR POLLUTION & HEALTH by R.E. Hester, R.M. Harrison